VentMonkey
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okidokie VentMonkey, no problem sorry I misinterpreted.
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okidokie VentMonkey, no problem sorry I misinterpreted.
These are just my thoughts, Nova. Feel free to chime in with yours.I waffle a lot on this question. On the whole, I'm not convinced prehospital intubation does much to save lives. Anecdotally, there are likely cases in which it helps. But to play it out statistically, it probably makes little mortality difference in the grand scheme.
That being said, I have always suspected that brain injured patients may be the one population in which there could be a benefit. I will tell you, a large number of these patients aspirate. You may not see it happen, but their bronchs consistently look awful on hospital day 1.
I have to wonder a couple things:
1. Does protecting the airway with a balloon, prior to strapping these patients supine and moving them around, decrease the incidence of these aspirations?
Would this be a patient population to benefit from early gastric tube placement in the prehospital setting?
2. Does the sedation provided for intubation in the field provide a protective effect to the injured brain by decreasing the cerebral metabolic demand during a time of great stimulation( lifting, moving and transporting)?
In my opinion said patients may benefit from a longer acting paralytic, or continued paralysis to furhter prolong any increases in metabolic demand/ ICP.
3. Does early control of respiratory acid/base status improve outcomes? Specifically when instituted prior to moving and transporting the patient?
Good question, and I don't have any definitive answers or proof, but will say that a fair amount of our "scene" patients are of the TBI variety, and furthermore, yet another reason PROPER prehospital ventilator management MUST be included when learning advanced airway management, and by advanced, I am referring to most of what is taught in the (United States') paramedic curriculum; again, simply "just getting the tube" does nothing for their recovery/ mortality ratio but prove our incompetence.
Perhaps these things would only have a benefit for longer transport times? Who knows. But this has always been a nagging question in the back of my mind. Because I have a hunch early intubation in these folks, specifically before transport, really could be of benefit.
@VentMonkey i agree with you. Ive learned alot of applicable information when i learned about ventilator management and it has helped me become a better clinician even without the vent. Im also a big fan of resuscitate before you intubate and that placing that ET tube alone is not a treatment. i also feel that not all RSI is created equal. The patient mentioned above should have a different RSI then the septic patient, asthmatic, CHFer ETC. and thus the medications, dosage, pre and post treatment should differ and maybe prehospital intubation would have better outcomes.
@TXmed, good point, and to further elaborate on the last part of @Nova1300's post, my personal opinion is that not all providers are created equal, and hence those properly trained for such specific situations should probably be the only ones coming near their airway with an advanced approach in the prehospital setting.early intubation in these folks, specifically before transport, really could be of benefit.
Yes, but I did this recently and caught all kinda of crap for it as well, sooo...am I in the wrong thread?
Unless you're referring to the earlier post re: treatment of the bradycardia, nonetheless, it sure is easy to ger things crossed.Incidentally in the past I was taken to task for giving lidocaine to a bradycardic patient (completely different bigeminy scenario). Obviously if the patient happens to be in a bi/trifasicular block lidocaine is bad, but I don't really see sinus bradycardia as an issue for premedicating with lidocaine, am I wrong?
I don't really see sinus bradycardia as an issue for premedicating with lidocaine, am I wrong?
Some people see bradycardia as a contraindication to lidocaine. I am not sure why.Unless you're referring to the earlier post re: treatment of the bradycardia, nonetheless, it sure is easy to ger things crossed.
And nah, I would equate it to the prophylactic treatment of bradycardia with Atropine, mainly in the pediatric RSI population, though, many places are moving away from that in their treatment tree as well TMK.
I can honestly say I don't recall learning this; guess it's time someone revisits this med, huh?Some people see bradycardia as a contraindication to lidocaine. I am not sure why.
32 mmHg. We ended up turning the O2 off and just BVMing him through his periods of apnea with room air. SpO2 stayed at 99-100%.
Nice job with the ETCO2 threshold. I am unfamiliar with the second part of this quote though, what's the theory?
I guess I'm going to be an outlier on this then. Given the 6 minute transport time to a L2 TC, I'm going to forego the intubation and simply BVM this patient. While I am a Paramedic and still remember how to intubate, I'm not above using my basic skills when they're quite appropriate to use and this is one such case. I'd just call ahead, give the radio report, and request that they be ready to do an RSI asap upon arrival due to trismus/Cushing's response. They need time to get a room ready for this and time to clear out a CT scanner for this patient.
Taking the time to RSI this patient on scene just eats up time that can be used getting him to definitive Neuro care and have them be ready to mange this guy's airway.
This is just one of those times when an ALS assessment results in BLS care getting the patient to definitive care in an expedient manner, but wouldn't change the outcome much.