How high is too high of a potassium to treat it with calcium chloride? I don't think I've seen the ER do dialysis during an arrest.
I am not knowledgeable on catherization during cardiac arrest. Do most STEMI facilities do this or just some? While the the arrest may have been caused by the occlude MI, it is possible to treat the arrhythmia without having to treat for the MI further? I know it wouldn't work in all cases, and sometimes an arrhythmia isn't the cause of the arrest from an MI, but wouldn't that work in some cases? Would it be reasonable to work the patient for 20+ minutes before making the decision to transport, if the cause is an MI?
I wouldn’t expect for emergent HD or CRRT to be imitated during arrest, however what methods are available on 911 ambulances to check a basic chemistry?
We cannot correct hyper K simply by giving calcium. In fact if you give too much calcium and we can’t pull off the K quick enough then you end up with irreversible contracture of the myocardium.
More importantly the function of potassium isn’t just relative to calcium outside the cell but also to the level of potassium inside the cell. Giving more calcium does not change that. We can temporize patients by things like continuous albuterol or giving insulin and dextrose both of which are not available to the vast majority of 911 providers.
If you recover an arrest without addressing the underlying pathology the chance of rearresting is essentially guaranteed. The hyperkalemic is likely to arrest in the bus again once en route and then the point of working on scene becomes moot.
Reperfusion codes are common the the cath lab during and after procedures, working a code in the cath lab isn’t uncommon or particularly difficult.
We certainly wouldn’t take a code to the cath lab without knowing that they need an appropriate intervention whether that be Dilation/stenting, placing a pacer, placing impellas, or whatever else.
While a large number of adult arrests are the result of MI we can’t take every undifferentiated arrest to the cath lab. The prevalence of MI in cardiac arrest is the largest driver of why we immediately get 12 lead if we get ROSC. Time is muscle, and a MI that has caused enough damage and irritability to cause ectopy leading to a fatal rhythm is extremely time dependent.
If a patient is known to have a high risk cardiac history like multiple prior stents and/or CABGs with a witness stating he reported chest pain or other associated symptoms and had a witnessed arrest with immediate bystander CPR then I would hedge on immediate transport if you have a cath lab within 30 minutes or so.
This is all what I was alluding to with my original post though. Many arrests are not viable and should not be transported. Some will need rapid transport to a major center for any chance at survival. The move for systems to make working codes until ROSC or presuming death on scene removes the clinicians decision making and is something I find particularly disturbing. It seems to be another trend in removing the paramedic scope of practice (like with intubation or RSI) rather than increasing education so that those decisions can be made.