- 9,736
- 1,174
- 113
So had a call today during my internship.
VT with pulses, pt was A&O, walking, talking, joking around, not really complaining of anything other than mild SOB and a "knot" in his throat.
Not really relevant though. My question is why is it that the diastolic pressure stays high while the systolic drops causing a very narrow pulse pressure.
The last pressure I saw before they sedated and cardioverted him in the ED was 121/106. Original pressure on scene was 178/90.
My undereducated and sleep deprived guess is that vascular tone is still intact therefore keeping a relatively constant diastolic pressure? Whereas the cardiac output is compromised due to the fast ventricular rhythm, inadequate ventricular filling along with the loss of atrial kick. The drop in CO causes the changes in systolic blood pressure, correct?
Sorry if this doesn't make any sense, I'm beat from today.
VT with pulses, pt was A&O, walking, talking, joking around, not really complaining of anything other than mild SOB and a "knot" in his throat.
Not really relevant though. My question is why is it that the diastolic pressure stays high while the systolic drops causing a very narrow pulse pressure.
The last pressure I saw before they sedated and cardioverted him in the ED was 121/106. Original pressure on scene was 178/90.
My undereducated and sleep deprived guess is that vascular tone is still intact therefore keeping a relatively constant diastolic pressure? Whereas the cardiac output is compromised due to the fast ventricular rhythm, inadequate ventricular filling along with the loss of atrial kick. The drop in CO causes the changes in systolic blood pressure, correct?
Sorry if this doesn't make any sense, I'm beat from today.