Cushing's triad

well i had a bunch of questions but ill ignore the big write up for this. how do you know this person is oxygenated well?

also everybody make sure you dont mix up hyperventilate and hyperoxygenate. not saying anyone has but i see it often. (pet peeve)
 
well i had a bunch of questions but ill ignore the big write up for this. how do you know this person is oxygenated well?

also everybody make sure you dont mix up hyperventilate and hyperoxygenate. not saying anyone has but i see it often. (pet peeve)

Many factors

Persons:

Color, respiratory rate and effort, spo2(not always reliable but its a tool), LOC, etc.

If the person is showing true Cushings then they will have irregular breathing. When I see that, then at my level and protocols that is my A and I will secure and airway and provide supplemental O2. How much will depend on the case and what I deem necessary.
 
color, this person will be pale well oxygenated or not. he will be shunting blood. if you mean cyanotic then yes i agree bag.

spo2 may read 97 percent but what is the body doing to acheive this, how much is it compensating and how is this affecting the vitals? what about secondary injuries, this is a very real possibility. hemmorhage?tamponade?pulm injuries? how are these affecting your "triad"

LOC- nobody will be mentating properly here so does that indicate NRB or BVM? what will the effect of this be? hyperoxygenation or hypocapnea?

irregular breaths- irregular doesnt necessarily mean ineffective and these pt's can had very strange breathing patterns that seem great than 10 seconds later then really shallow/deep or tachy/brady. whats acceptable?

those are some of my questions... I go NRB on these pt's (head injuries, dont really see the whole triad very often) and wait for signs that i need to bag. traumas dont seem to need it as often as strokes do for me..funny now that i think about it
 
color, this person will be pale well oxygenated or not. he will be shunting blood. if you mean cyanotic then yes i agree bag.

1.) spo2 may read 97 percent but what is the body doing to acheive this, how much is it compensating and how is this affecting the vitals? 2.) what about secondary injuries, this is a very real possibility. hemmorhage?tamponade?pulm injuries? how are these affecting your "triad"

3.) LOC- nobody will be mentating properly here so does that indicate NRB or BVM? what will the effect of this be? hyperoxygenation or hypocapnea?

4.) irregular breaths- irregular doesnt necessarily mean ineffective and these pt's can had very strange breathing patterns that seem great than 10 seconds later then really shallow/deep or tachy/brady. whats acceptable?

those are some of my questions... I go NRB on these pt's (head injuries, dont really see the whole triad very often) and wait for signs that i need to bag. traumas dont seem to need it as often as strokes do for me..funny now that i think about it

Those are some excellent questions!

1.) SpO2 can be the devil! It is a great tool but not without it's flaws. Perfect example was a pt of mine yesterday. Pt presented w/ respiratory distress- respirations 28 labored and bi-lateral lungs presented w/ rales (hx CHF). SpO2 read 92%....

The pt presented w/ a sympathetic response. Sinus tach around 110 (pt was also on a beta blocker), skin ashen in color w/ pale conjunctiva and mucosa, cool, slight diaphoresis, and cyanosis was noted to her fingertips. The pt's increased respiratory rate and exertion were maintaining the O2 saturation but a pt can only do that for so long....

Yes, shock will throw vitals askew, especially if you go in expecting to see vitals associated w/ a certain condition (ex- Cushings Triad). If a pt has head trauma think ICP, regardless of the vitals.

2.) Secondary injuries also will debunk any predetermined diagnosis based on "syndromes" that rely on a vital sign like Cushings or Becks. Most pt's w/ significant head trauma or thoracic trauma w/ be treated as multi-system trauma and present as such.

3.) If a pt is unconscious/unresponsive an airway w/ assisted ventilations will be provided. A BLS provider should insert a basic adjunct and assist ventilations if the unconscious/unresponsive pt presents w/ respiratory distress or respiratory failure. The pt will be hyper-oxygenated due to the supplemental O2 but that's the point. To be ahead of the 8 ball. Hypocapnia can only be determined by pre-hospital by ETCO2. If you hyperventilate a pt they will be hypocapnic. Obvious signs of brain herniation should be the only time this method of assisted ventilations should be utilized in a attempt to reduce or "slow down" ICP. Medical control should be contacted before preformed unless a protocol is in place.

4.) Irregular or inadequate is not acceptable. Every pt is different and if the pt appears to be experiencing inadequate tissue perfusion from such, then assisted ventilations should be considered/preformed.
 
So as a basic... Can I really worry about a patient becoming hypocapnic? I mean I've never even been taught to worry about that yet. lol i wasn't even sure what it meant. But I'm assuming too much or too little co2?

I was taught to never withhold o2 from anyone that needs it. Even with a chronic COPD. Although giving them too much o2 can shut down their respiratory drive. If they are turning blue and not adequately perfusing then I would give o2 via a NRB.

And as I stated before in my little disclaimer....spo2 is just a tool and not always reliable.

Any pt who becomes unconscious is considered to not be able to maintain their own airway. So they would get an OPA and maybe BVM depending on the situation.

Someone who is conscious will either get an NPA (if there is no trauma) and they will for sure get o2 in some form or another and possibly the BVM. Untill I can get them to ALS or the hospital. Either or.

That's what I know how to do. So I am going with that.

Am I wrong?
 
angel, i am a paramedic student right now so i bounce around between the the basic and advanced forums. you are right that you shouldnt really have to worry about hypo (HYPO means......)capnea but it is a valuable concept to understand at any level. take a bit of time to look it up and oxygen/hemoglobin dissociation curve.

id assume u dont have any protocols involving bagging a head injured patient with supected impending brainstem herniation so dont worry about that.

your treatment is spot on. the only thing i might want to add is that the whloe COPD/respiratory drive thing is (in my opinion) overstated. It can happen but doesnt often, that said dont listen to some guy on the internet vs what you have been taught by a text and your teachers.

and to J. Burdett... i agree 100% with your post. i meant to state my questions as more of an "ask yourself" type question but your reply ended up giving me a great read. thanks
 
As a basic.

What is the most appropriate pre hospital care for someone you suspect to be exhibiting the signs of the Cushing's triad?

I am writing a paper and would like some of your thoughts.

If an ALS peeps see this and want to respond with what they would do, then by all means go ahead.

THANKS! B)

p.s I hope i put this in the right category haha

Drive quickly to somewhere with neurosurgical capability.
 
remember this when considering increased ICP- hyperventilating a pt with increased ICP will lower thier PaCO2 and cause cerebral vasoconstriction, thus minimize brain swelling- which we want. This is an important intervention as a BLS provider. Also, if it is in youre protocol, monitor ECG along with (obviously) Sp02.
'Cushings relex' will present only in the early stages of increased ICP. More sever ICP will present with compensatory vital signs. +HR, +resps, -BP (opposite of cushings reflex)
 
remember this when considering increased ICP- hyperventilating a pt with increased ICP will lower thier PaCO2 and cause cerebral vasoconstriction, thus minimize brain swelling- which we want. This is an important intervention as a BLS provider. Also, if it is in youre protocol, monitor ECG along with (obviously) Sp02.
'Cushings relex' will present only in the early stages of increased ICP. More sever ICP will present with compensatory vital signs. +HR, +resps, -BP (opposite of cushings reflex)

Hyperventilation is pretty controversial as to whether it helps or hurts.

I think your confused, Cushings Triad is a very late sign, i.e. herniation. What you see next is the vital signs go absoloutely haywire (massively increased or decreased HR, extreme hypertension, very very odd depressed respiratory patterns) followed a couple of minutes later by the absence if vital signs.
 
Hyperventilation is pretty controversial as to whether it helps or hurts.

I think your confused, Cushings Triad is a very late sign, i.e. herniation. What you see next is the vital signs go absoloutely haywire (massively increased or decreased HR, extreme hypertension, very very odd depressed respiratory patterns) followed a couple of minutes later by the absence if vital signs.

ha ok this is what I thought. I was starting to second guess myself for a minute.

Diesel.



^_^

HAHAHAHA pretty much. Everything I have read about this is you're damned if you do and damned if you dont. So PUHA!
 
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