Protoman2050
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Here's an interesting scenario. Reason I'm asking this is b/c I'm a CVT student, and I keep imagining I find myself in this scenario. I hope I don't!
Scenario:
A cardiovascular technologist who contracts with the local hospital and uses his own equipment, is eating his lunch on a park bench. He is chatting with a middle-aged man, who suddenly starts having angina and dyspnea. He is sweating, his skin is cool to the touch and pale, and his jugular veins are distended.
The CVT activates EMS, gets in contact with medical control, and asks for an ambulance and permission to perform an 18-lead EKG and a transthoracic echocardiogram.
You, a paramedic, arrive to the site, and the CVT shares his findings with you:
Symptoms and physical findings: Angina and dyspnea. He has slight wet rales, and he has a holosystolic murmur from S1 to S2. Patient also has severe jugular vein distension.
VS: BP: 90/50, RR: 18, HR: 100 bpm, Temp: 37 degC
EKG: 18-lead EKG shows large Q-waves and 5 mm of ST elevation in leads II, III, avF, V1-3, V3R, and V4R. Afib and PVCs are also noted.
ECHO findings: Severe acute MR due to torn papillaries causing flail leaflets. Severely hypokinetic left and right inferior wall. Mean pulmonary artery pressure is 6 mmHg.
He says that the pulmonary edema is less then he thought it would be, but that's only because the diminished LV preload isn't giving much for the MV to regurgitate into the lungs.
You activate the cardiac cath lab, and page the on-call cardiac surgeon at the local hospital.
How would you stabilize this patient?
Would you put the patient on CPAP, administer 0.5 mcg/kg/min of dobutamine and 0.5 mcg/kg/min of Nitropress to augment the patient's cardiac output and reduce the afterload of the ventricles.
How would you maintain LV preload without throwing the patient into FPE?
What would you do about the Afib and PVCs? 150 mg amiodarone over 10 minutes?
Would you start Activase to attempt to restore perfusion?
Thanks,
Doug
Scenario:
A cardiovascular technologist who contracts with the local hospital and uses his own equipment, is eating his lunch on a park bench. He is chatting with a middle-aged man, who suddenly starts having angina and dyspnea. He is sweating, his skin is cool to the touch and pale, and his jugular veins are distended.
The CVT activates EMS, gets in contact with medical control, and asks for an ambulance and permission to perform an 18-lead EKG and a transthoracic echocardiogram.
You, a paramedic, arrive to the site, and the CVT shares his findings with you:
Symptoms and physical findings: Angina and dyspnea. He has slight wet rales, and he has a holosystolic murmur from S1 to S2. Patient also has severe jugular vein distension.
VS: BP: 90/50, RR: 18, HR: 100 bpm, Temp: 37 degC
EKG: 18-lead EKG shows large Q-waves and 5 mm of ST elevation in leads II, III, avF, V1-3, V3R, and V4R. Afib and PVCs are also noted.
ECHO findings: Severe acute MR due to torn papillaries causing flail leaflets. Severely hypokinetic left and right inferior wall. Mean pulmonary artery pressure is 6 mmHg.
He says that the pulmonary edema is less then he thought it would be, but that's only because the diminished LV preload isn't giving much for the MV to regurgitate into the lungs.
You activate the cardiac cath lab, and page the on-call cardiac surgeon at the local hospital.
How would you stabilize this patient?
Would you put the patient on CPAP, administer 0.5 mcg/kg/min of dobutamine and 0.5 mcg/kg/min of Nitropress to augment the patient's cardiac output and reduce the afterload of the ventricles.
How would you maintain LV preload without throwing the patient into FPE?
What would you do about the Afib and PVCs? 150 mg amiodarone over 10 minutes?
Would you start Activase to attempt to restore perfusion?
Thanks,
Doug