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The most important one to know (by far) is hyperkalemia.
The progression is from peaked T waves to a flattening of the P wave, widening of the QRS complex, and a merging together of the S wave and T wave (so-called sine wave ECG).
The history is usually significant for renal disease and the classic presentation is a missed dialysis appointment.
The sine wave ECG is sometimes referred to as "slow VT" since P waves are often absent by that time but the rate is < 100.
Anytime you have a QRS complex that approaches 200 ms you should be very suspicious of hyperkalemia.
Type 'hyperkalemia ecg' into the image search engine at Google to see some examples.
Tom
46,
Let me know how to e-mail you a flash chart. You'll love it.
I've seen instances of attempted TCP with hyperkalemia, but never with capture (of course I could say that with almost every TCP case I've ever reviewed, whether they were hyperkalemic or not).
Funny you should mention that, we had a hyperkalemic pt the other day. The pt was alternating between a widecomplex brad and asystole. TCP captured @ 60BPM, and 89 milliamps. It worked quite well till we arrived @ the hospital and the pt was given the bicarb, ventolin, calcium chloride combo, within 10 min he was mentating normally and in normal sinus @ 70 BPM.