Acute hypoxic event mimicking closed head injury?

Aidey

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From the research I've done it sounds like it is possible to have posturing from an acute hypoxic event or anoxic brain injury. But I am still a little fuzzy on how similar the presentation to a closed head injury would be, and if it would be possible to tell them apart. And if the symptoms would resolve once the hypoxia was corrected.

Here is the situation. The patient was assaulted, and at the time we had no idea what had happened. That was all the info we had. My judgment at the time was that he had a severe closed head injury. We found out later that he did not have a closed head injury, but he had been strangled into unconsciousness.

This was his presentation, the pt is on a back board, with a c collar on, and someone holding manual c-spine since he wasn't fully strapped down.

Neuro: GCS of 6? (we think, see below)

The patient's initial BP is 150/80 with a pulse of 80. Second BP is 170/100 with a pulse of 54. SpO2 is reading 68% on a mask, however the pt didn't have generalized cyanosis. He had so much blood on his face it was impossible to tell the color of his lips, but I don't believe his SpO2 was that low.

There was no change in his presentation when we talked to him, and he was not following commands. Lower extremities have decorticate posturing and he isn't moving them. He is flailing his arms around but isn't making a fist or trying to grab anything, so as far as we can tell, no purposeful movement. He is grunting occasionally. His eyes are totally swollen shut, with dark bruising around both. The edema so so bad it is difficult to make out the margins of the orbit. There blood leaking out of both eyes. It was impossible to assess eye response for the GCS, so I went with the worst case and gave him a 1.

Anyway, we find out later he had been strangled, and he had no closed head injury or bleeding at all. The eye swelling was from direct trauma to the eyes. As far as I can find out, he was AOx4 the next day, without any obvious deficits.
 
How was his breathing? If that was changing along with the given BP and HR, I'd have Cushing's triad on my mind as well.
 
Oh yeah, that. lol. Sorry, my post wasn't very organized. It was fast and shallow, ~30 per min or so.
 
Remember that whether the mechanisim is strangulation, closed head injury, cardiac arrest or a failed airway it's all cellular hypoxia, so many of the signs and symptoms will be similar.
 
Remember that whether the mechanisim is strangulation, closed head injury, cardiac arrest or a failed airway it's all cellular hypoxia, so many of the signs and symptoms will be similar.

That is pretty much my take on it too, the mechanisim is not important really as the underlying physiologic abnormality is all the same.

Now, where is that RSI trained Intensive Care Paramedic or Doctor I requested?
 
I did RSI him, and I don't even have DOCTOR on my jumpsuit. :P

I get that hypoxia is hypoxia, regardless of cause. It is the presentation vs how fast the presentation resolved that has me confused. So I guess the real question is that with proper oxygenation can the hypoxia be reversed, thus resolving the symptoms without any (major) long term damage occurring?
 
I did RSI him, and I don't even have DOCTOR on my jumpsuit. :P

I get that hypoxia is hypoxia, regardless of cause. It is the presentation vs how fast the presentation resolved that has me confused. So I guess the real question is that with proper oxygenation can the hypoxia be reversed, thus resolving the symptoms without any (major) long term damage occurring?

Given the timeframe and severity of hypoxia, while considering comorbid factors, the short answer could be yes?
 
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I guess so ....

Oh and our Intensive Care Paramedics (ALS) have RSI now, I think the PRIME Doctors (like BASICS in the UK, sort of) have had for about the same length of time.

Brown finds strangulation too messy and time consuming, so he just carries round a large beheading sith :D
 
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