It all started with Oxyhemoglobin Dissociation Curve theories and my judgment on those. I apologize, I am sorry. But I just wanted to convey that a absolute good PSO2 does not correspond to tissue profusion. The topic got twisted to MI's which was way off topic. But I am good with that too.
And you should never have taken issue with treat the patient not the monitor, it is a rule taught in every med school, nursing school, and paramedic class ever held. Or it should have been, if your instructor was worth anything.
It is good your take issue with it, so honestly as a clinician what would you believe?
Your own clinical judgment or that machine? that is all I stated, and yet you got all pissy?
Tell me?
Actually lets just agree to disagree okay. Because by definition a STEMI is an ST elevated MI. But a normal 12 lead does not rule out an MI, but it does rule out a STEMI. There are more than one MI's and the treatment rules dictate various approaches, such as sub endocardial just to name...
A old EMS axiom, watch your own *** first, or there will be no one around to treat the patient.
Do not try and out hero the other person, that is only for TV.
Sorry, I did not mean to jump your ***. but until you learn the difference between compensated and uncompensated distress and know the values of oxy-hemoglobin curves then you will not understand the effects of resp distress on a patient. Your point is valid on non typical acute cardiac...