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Just looking for some thoughts on this call the other day.

Discussion in 'Scenarios' started by RanchoEMT, Mar 12, 2012.

  1. mycrofft

    mycrofft Still crazy but elsewhere

    Central California

    Seriously, with the trend now to train every layperson to address adult loss of consciousness with (at least) hands-only CPR, we need to start seeing postmortems recording if the CPR we teach causes signifcant cardiac insult to offset benefit. Used to be starting CPR when any pulse was palpable was a "horror of horors".
  2. Steveb

    Steveb New Member

    At times I don't even know
    EMS Training:
    What is a PE???
  3. SoCal911

    SoCal911 New Member

    Pulmonary embolism
  4. Maine iac

    Maine iac New Member

    Halfway through reading this my idea was PE, then it changed to a witnessed AAA, and now I am some what torn between the two (hahah torn.. :rofl:). He was playing racket ball earlier.. might have gotten hit in the chest causing a dislodge of plaque going into his lung causing a chain reaction and death.

    I would be interested to know what the FAST exam showed. If he was still in PEA at the hospital, doubtful, but I would hope that the hospital would be aggressive to try everything for him.

    Did you guys mess around on the scene or was this like a 4 minute scene time? Not that it would have changed anything at all- but just wondering.
  5. exodus

    exodus Active Member

    Hemet, Ca
    Do you have the 12 lead still? I'd like to see it.
  6. RanchoEMT

    RanchoEMT New Member

    I'll get it when i go back to work, thursday...
  7. RanchoEMT

    RanchoEMT New Member

    you know what, honestly, they didnt try for very long. Me and my medic where kinda hopefull... We both were kinda bummed afterwards cus it seemed like they didnt try as hard as they could have for a witnesssed arrest like this.

    Scene time was prolly 10-15 minutes... 45 seconds getting to patient, patient coded about 5 min after, we worked on him for about 7 minutes, then transported. 10-15ish...
  8. mycrofft

    mycrofft Still crazy but elsewhere

    Central California
    Never heard of blunt force dislodging plaque. Those are in the vessel wall, not like grease in the drain trap like we initially thoght.
    But blunt force could cause cardiac tamponade (see above).
    Last edited by a moderator: Mar 14, 2012
  9. Squad51

    Squad51 New Member

    Echo echo echo. I was just thinking the same thing about the hernia but you beat me to it! I agree with everyone else. Most likely a PE. Depending on the location of the clot, could be why he was so tachy and hypotensive with decreased SpO2. The clot probably dislodged and put him in full blown cardiac arrest. Not much you could do different. The "mass" probably had nothing to do with it. He probably had a hernia that he did or did not know about. Certainly fits the description you gave when you sat him up.
  10. RanchoEMT

    RanchoEMT New Member

    Lets say for a second it was an aneurysm and the patient did go into a PEA full arrest. Is it possible(and/or probable) that the PEA showing on the monitor and verified by a lack of palpaple pulse was in fact Wrong??? Again, Assuming for a second that it was in fact an aneurysm is it possible that the heart was still beating, but due to the aneurysm, the distant pulses were not pulsating?

    Obviously, the heart bleeding out wouldn't last for long with the drop in blood volume, (resulting in a true arrest) but Does any one auscultate the heart in a full arrest just to verify this???

    ***Noticed, when they called him later at the hospital, he was still in "PEA". ***
  11. 94H

    94H New Member

    EMS Training:
    Im thinking AAA also just because of the mass. Did you notice if it was pulsating at all?
  12. jjesusfreak01

    jjesusfreak01 New Member

    St. Georges, Grenada
    EMS Training:
    I don't see how they got a decent SpO2 on this patient to begin with, considering the lack of peripheral pulses, unobtainable pressure, and obvious shunting away from the periphery.
  13. TheLocalMedic

    TheLocalMedic Forum Asst. Squirrel

    Not an AAA, but rather a rupturing thoracic aneurysm. With a thoracic aneurysm it is not uncommon to get upper limb cyanosis, just like an AAA can cause lower limb cyanosis. The difficulty breathing sounds like referred pain and an attempt to "self splint" by decreasing chest wall movement. I had a patient a few years ago that exactly matches the description of this patient and a postmortem revealed an aneurism that began at the aortic arch and descended past the diaphragm.

    And who was it earlier that questioned the use of high flow O2? Really? When a patient looks like crap, give them the extra fuel. The whole issue of using lower amounts of O2 pertains mainly to prolonged use and stable ACS or COPD patients, remember that you should never withhold O2 if they're really sick. You don't look smart when you do that, it just shows that you haven't truly read the research about what you're preaching.
  14. usalsfyre

    usalsfyre You have my stapler

    Right, high flow O2 makes all the difference for hypemic issues because the oxygen carrying capacity of plasma is so great :rolleyes:...

    If you truly have a ruptured abdominal aneurysm to the point of exangunation you'd likely only have a distended, rigid abdomen due to the amount of peritoneal irritation. A ruptured thoracic aneurysm is more likely but my money is on PE.
    Last edited by a moderator: Apr 11, 2012
  15. Handsome Robb

    Handsome Robb Youngin' Premium Member

    EMS Training:
    I still want to see the 12-lead :D

    As far as listening for an apical pulse you could but if you can't find a carotid pulse the guy isn't perusing his brain so I doubt it's going to matter much. Might be viable for organ donation but with the shunting I'm not sure the organs would be viable anyways...
  16. RanchoEMT

    RanchoEMT New Member

    I looked for it when i came back to work and realized fire used their monitor, its saved on their LP15... In other words its gone, sorry.
  17. leoemt

    leoemt New Member

    Washington State
    EMS Training:
    I know this thread is a couple of months old so don't yell at me for reviving it but I am really interested in this scenario.

    While I don't have the experience yet to really offer a guess I have noticed that no one has keyed on the fact that the patient was playing Racket ball prior to onset of symptoms.

    Is it possible that the patient took a hit to the chest by the ball? That would be enough of blunt force trauma to cause a pericardial tamponade (sp?) I would think.

    Having played racket ball myself I know those hard rubber balls can really get moving. I've seen players get broken bones from those balls.
  18. TB 3541

    TB 3541 New Member

    Inland Empire
    EMS Training:
    Actually, someone did bring that up:

    I'm really intrigued by this scenario too, but unfortunately, I don't think we will ever know because we don't have access to any records other than the first-hand experience provided. Really, I think only an autopsy could be definitive.
  19. blindsideflank

    blindsideflank New Member

    Problems with coarctation of aorta/dissection
    Saddle embolus?

    Murder and a fake story
    Last edited by a moderator: Jul 18, 2012
  20. Chase

    Chase Murse

    Saint Louis, MO
    Was he in chronic A fib or was this new onset? Any current medications, particularly any Anticoags? Not with this situation but with symptoms after physical activities it always keep spontaneous pnuemo --> tension pneumo in the back if my mind.

    Blue from the clavicles up....Line of demarcation?
    Last edited by a moderator: Jul 18, 2012

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