With all the research thats going on in Durham at DUKE, a couple of docs have started research on high flow 02 in pts with embolic CVAs. Their argument is, in the time it takes to initiate treatment and transport of the patient, the high-flow oxygen that we are used to providing actually causes capillary/vasoconstriction which may worsen the effects of these emboli. Anybody heard anything comparable in their neck of the woods? A lot of medics here are starting to put pts on NCs with acute embolic CVAs as long as they are portecting their airways. Any thoughts?
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