teedubbyaw
Forum Deputy Chief
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Not sure why this concept isn't sinking in...
What are the mechanisms of a drug that works at the AV junction (adenosine, diltiazem, etc. etc.) and it causing an increased rate in WPW? I can understand why it would be an issue in orthodromic, but what about antidromic?
What are the mechanisms of a drug that works at the AV junction (adenosine, diltiazem, etc. etc.) and it causing an increased rate in WPW? I can understand why it would be an issue in orthodromic, but what about antidromic?