with regards to the body's compensating mechanisms for hypoperfusion...
increased HR, peripheral vasoconstriction, cool clammy skin, increased RR, etc...
with regards to the BP... my understanding is that it is the CO X Peripheral resistance (afterload)... as the CO drops, the body tries to compensate by increasing the afterload... basically, the body's compensating mechanism tries to maintain it's BP...
if my understanding of this is crude, but correct, does it explain why BP is not always a reliable Vital Sign for compensated shock?
increased HR, peripheral vasoconstriction, cool clammy skin, increased RR, etc...
with regards to the BP... my understanding is that it is the CO X Peripheral resistance (afterload)... as the CO drops, the body tries to compensate by increasing the afterload... basically, the body's compensating mechanism tries to maintain it's BP...
if my understanding of this is crude, but correct, does it explain why BP is not always a reliable Vital Sign for compensated shock?