As already stated, there are usually prominent ways to tell them apart.
If someone is hypoglycemic, the signs and symtpoms are pretty obvious. Either the patient will know they need sugar and will tell you how they feel, or they will be in an obviously altered mental state/unconscious. Many of these patients also come with pretty profuse diaphoresis + cool skin.(sweating)
If the patient is with a group, ask them for some background info. Hypoglycemia doesn't just hit you like a brick and you collapse. It is a gradual burning up of the fuel for your brain.
If a patient presents with an altered mental state, odds are they are NOT hyperglycemic. People with high sugar lack the ability a normal person does to either
A: Produce insulin
B: Utilize insulin properly.
C: Combination of A and B
This is where either a pill to aid in the facilitation of insulin/glucose use comes in. Or actual insulin injections.
A patients medications can tell you an awful lot about them if you know what to look for.
To name a few:
Insulin - Humalog, Novalog, Humulin, Lantus
Pills - (Sitagliptin) Januvia, Glucophage (metformin)
A type one diabetic will always take insulin because there pancreas does not produce it.
A type two diabetic will usually take a pill, sometimes insulin as well because their body cannot produce sufficient amounts of insulin at the same time as having problems utilizing it.
Anyway, a person with hyperglycemia will rarely present with an altered mental state unless they have been hyperglycemic for a while. And by a while, I mean days, sometimes even longer. It is very rare someone be symptomatic with hypergylcemia in an acute setting.
That said, the damage from hyperglycemia does not come from having too much sugar in your system. The damage comes from what your body does in place of the sugar. Since the glucose in your blood cannot get into the cells without insulin, it simply stays there and your blood sugar will just continue to rise no matter how much you eat. Your body now needs to find an alternative source of fuel that it can use without insulin. The body turns to fat. It burns fatty acids in effort to keep the engine running. The problem with fatty acids, is that burning them produces ketone bodies which are acidic. After a long enough timeframe the buildup of ketone bodies will cause the pH of the body (your body is similar to a pool) to become too acidic for normal physiologic function to continue. The body needs to maintain a nearly neutral pH. (its actually very slightly basic) So with this low pH (acidic) the body tends to try and bring balance every way it can. This usually causes the patient increased urination in an effort of the kidneys to remove acids from the body. With that excess loss of fluid from urination (polyuria), dehydration sets in, which results in increased thirst.(polydipsia) (which will reflect via the skin and mucus membranes as well)
Little side note, the brain continues to function relatively normally during hyperglycemia because the brain can work off of "serum" glucose. This means that for primary functions it really doesn't NEED insulin. And without the rest of the body using the sugar up, the brain has plenty to enjoy.
So, in short, giving sugar to a hyperglycemic patient, in such low doses, is not going to have any harmful effects. Worst case it just raises their blood glucose a little more, but that isn't what is causing the real damage to their body. Also if someone is in a severely dehydrated state with hyperglycemia, giving them something by mouth can sometimes cause nausea.
Don't quote me to the letter on any of this, It sounds right in my head but im sure some of the forum brain surgeons will chime in at some point
